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Atherosclerosis progression, as a chronic inflammatory mechanism, is characterized by immune system dysregulation associated with increased pro-inflammatory cytokine production, including interleukin 6 (IL-6), tumor necrosis factor- (TNF-), and IL-1 (Vinciguerra et al., 2020
Mihm et al, 1999), is involved in the oxidant stress-mediated regulation of numerous gene expression, particularly at the transcriptional level
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We computed the ratio of the projected area to the nostril area, as a percentage
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Redox regulation of antioxidants, autophagy, and the response to stress: implications for electrophile therapeutics
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